Researchers in the United States have shown that genes from severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) - the causative agent of coronavirus disease 2019 (COVID-19) - can be integrated into the genome of infected human cells. The team says the viral RNA can be expressed as chimeric transcripts with fused cellular and viral sequences.

Australian scientists have demonstrated a new way to restore insulin production in pancreatic cells, using a drug that's already approved for use in humans. The study could mark a major breakthrough towards new treatments for diabetes. Blood glucose levels are managed by the hormone insulin, which is produced in beta cells in the pancreas.

After decades of inertial confinement fusion research, a record yield of more than 1.3 megajoules (MJ) from fusion reactions was achieved in the laboratory for the first time during an experiment at Lawrence Livermore National Laboratory's (LLNL) National Ignition Facility (NIF) on Aug. 8, 2021.

A shocking study from Israel exposes the coronavirus vaccine lies being pushed by the US government.

A new study has found that individuals that have previously contracted COVID-19 show a more potent antibody response than those who were solely vaccinated for the respiratory virus.

The SARS-CoV-2 virus has now become one of the greatest causes of infectious death and morbidity since the 1918 flu pandemic. Substantial and unprecedented progress has been made in the elucidation of the viral infection process in a short time; however, our understanding of the structure-function dynamics of the spike protein during the membrane fusion process and viral uptake remains incomplete.

Pitting COVID-19 drug candidates against the virus in cell cultures doesn't accurately predict how effective they'll be in the human respiratory tract. Now more accurate models of the human lung airway are uncovering new treatment candidates. They include the antimalarial drug amodiaquine, which reduced viral load in animal models of the virus by 70%.

SARS-CoV-2, the novel coronavirus infection has consistently shown an association with neurological anomalies in patients, in addition to its usual respiratory distress syndrome. Multi-organ dysfunctions including neurological sequelae during COVID-19 persist even after declining viral load.